Glutamine - A Medical Dictionary, Bibliography, and by Icon Health Publications

By Icon Health Publications

This can be a 3-in-1 reference ebook. It supplies a whole clinical dictionary masking thousands of phrases and expressions with regards to glutamine. It additionally offers vast lists of bibliographic citations. ultimately, it offers details to clients on how you can replace their wisdom utilizing a number of web assets. The e-book is designed for physicians, scientific scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to familiarize yourself with learn devoted to glutamine. in the event that your time is efficacious, this booklet is for you. First, you won't waste time looking out the web whereas lacking loads of appropriate details. moment, the e-book additionally saves you time indexing and defining entries. eventually, you won't waste time and cash printing thousands of websites.

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Extra resources for Glutamine - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References

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Generate_Screen • Project Title: MECHANISM CYTOTOXICITY OF ANDROGEN RECEPTOR INDUCED Principal Investigator & Institution: Lafevre-Bernt, Michelle; Buck Institute for Age Research Novato, Ca 94945 Timing: Fiscal Year 2003; Project Start 01-AUG-2003; Project End 31-JUL-2005 Summary: (provided by applicant): Kennedy's disease, or spinal and bulbar muscular atrophy (SBMA) is caused by an expansion of CAG repeats (which encode for glutamine) in the 5' end of the coding region of the androgen receptor (AR) gene.

Aim 3 will characterize the changes in Mrp2 expression in pregnancy and test the hypotheses that A) estrogens mediate the decreased hepatic Mrp2 expression in pregnancy, B) Mrp2 is subject to transcript-specific translational control conferred by regulatory elements in the 5' or 3' untranslated regions of Mrp2 mRNA and C) pregnancy and estrogens increase the degradation of Mpr2 protein. We will use polysomal distribution analysis of Mrp2 mRNA, translation assays in HepG2 cells and determine the degradation half-life of Mrp2 protein in control, pregnant, and estrogentreated rats.

MCH depresses glutamatergic and GABAergic synaptic transmission in the LH slices. 4. Characterization and organization of MCH responding neurons in LH slices will be documented. This proposal will begin to bridge the gap between molecular research and systemic studies on the regulation of feeding to benefit those suffering from obesity and obesity-related diseases. The applicant's previous work in characterizing the cellular physiology of the newly discovered neuropeptide hypocretin/orexin is the first step in his journey to pursue his long-term goal of understanding neurotransmission in the LH.

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